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  • Pathophysiological Mechanism of Bone Loss in Type 2 Diabetes Involves Inverse Regulation of Osteoblast Function by PGC-1alpha and Skeletal Muscle Atrogenes: AdipoR1 as a Potential Target for Reversing Diabetes-Induced Osteopenia.

Pathophysiological Mechanism of Bone Loss in Type 2 Diabetes Involves Inverse Regulation of Osteoblast Function by PGC-1alpha and Skeletal Muscle Atrogenes: AdipoR1 as a Potential Target for Reversing Diabetes-Induced Osteopenia.

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2015 Jul Others Fist_Author_Year_PM

Full Author : ['Khan, Mohd Parvez', 'Singh, Abhishek Kumar', 'Joharapurkar, Amit Arvind', 'Yadav, Manisha', 'Shree, Sonal', 'Kumar, Harish', 'Gurjar, Anagha', 'Mishra, Jay Sharan', 'Tiwari, Mahesh Chandra', 'Nagar, Geet Kumar', 'Kumar, Sudhir', 'Ramachandran, Ravishankar', 'Sharan, Anupam', 'Jain, Mukul Rameshchandra', 'Trivedi, Arun Kumar', 'Maurya, Rakesh', 'Godbole, Madan Madhav', 'Gayen, Jiaur Rahaman', 'Sanyal, Sabyasachi', 'Chattopadhyay, Naibedya']
Journal Title : Diabetes
Chapter : Section3
Journal Year : 2015
Cited by : Not Available

Type 2 diabetes is associated with increased fracture risk and delayed fracture healing; the underlying mechanism, however, remains poorly understood. We systematically investigated skeletal pathology in leptin receptor-deficient diabetic mice on a C57BLK.....

Keywords : ["type diabetes involve inverse regulation", "skeletal muscle atrogene", "reverse diabetesinduce osteopenia", "potential target", "pathophysiological mechanism", "osteoblast function", "bone loss"]